The Vulnerability of Acquiring a Lung Infection
“We must be vigilant as we are fighting to remain healthy one step at a time.”
We are going to see lung infections become much more prevalent because many times it is fungal based and antibiotics are becoming ineffective in treatment. It is not about lung infections being that much more dangerous but about the fact that so many people have become weaker and less resilient; and it is not by surprise they have predicted an increase for years.
People suffering from pain have risen dramatically, they have developed more inflammation which causes increased swelling and circulation problems accelerating cortisol dysregulation complex which leads to increased chronic stress and the perpetuation of pain. It becomes a medical treadmill as the various medications treat only the symptoms. The system will never tell you the truth, actually they could not even if they wanted as we are being poisoned on a daily bases on so many levels. It is mind-blowing how wide spread it has become with no control measures insight.
They will not change the antibiotic use in our food supply, they will not reduce toxic herbicides or pesticides, but they will offer predictions that the disease crisis will dramatically increase and that more people will die. If you are still under the illusion that the WHO, FDA, CDC and Health Canada are there for your improved health, then you should be fine since you will have increased vaccinations, multiple chemical drugs and surgeries—all part of getting old, at least that is what you will be told and need to believe.
The problem of antibiotic resistance
Antimicrobial resistance (AMR) has become a major global health threat, an estimated 4.95 million deaths were associated with it in 2019. While antibiotics are the primary drivers of AMR, evidence shows that nonantibiotic medications (NAMs) can also contribute.
Researchers at the University of South Australia investigated whether commonly prescribed NAMs could enhance antibiotic-induced mutagenesis in Escherichia coli. Nursing homes were chosen as a model because older adults often take multiple medications daily and are among the highest consumers of antibiotics, which are frequently overused in long-term care settings. According to their study Antimicrobials and Resistance[1] two widely used painkillers, ibuprofen and acetaminophen, significantly increased mutation rates and bacterial growth when combined with ciprofloxacin (an antibiotic used to treat bacterial infections), thus exacerbating antibiotic resistance. Across two E. coli strains, resistance extended to multiple antibiotics, with ciprofloxacin resistance increasing up to 32-fold, highlighting the role of nonantibiotic drugs in driving AMR.
Interestingly, not all pain management failed in the research. “The DMSO control (ciprofloxacin plus vehicle) consistently showed the lowest mutation frequency of all groups tested. None of the non-antibiotic medications (including ibuprofen, acetaminophen, diclofenac, or furosemide) reduced mutation rates below the level seen in the DMSO control; rather, several NAMs significantly *increased* ciprofloxacin-induced mutagenesis compared with that baseline.”[2]
This is why you really should avoid over-the-counter painkillers like ibuprofen and acetaminophen while you are on antibiotics because they enhance bacteria’s ability to resist antibiotics. Even better, avoid over the counter painkillers and rather try natural options. Also remember to use antibiotics only when it is absolutely necessary and avoid meat (the one found in grocery stores or restaurants) that may contain antibiotic residue.
Lung disease, lung cancer and what to do
Chronic obstructive pulmonary disease (COPD) commonly coexists with cardiovascular, metabolic, and psychological conditions that worsen quality of life and prognosis and lifestyle strategies become extremely important in this case. Both the obvious (stop smoking) and less obvious choices (nutrition) play a huge role alongside medical and pulmonary therapies.
It is not just about focusing on single nutrients; experts emphasize overall dietary patterns, with the Mediterranean diet as the best-supported option. This diet emphasizes fruits, vegetables, whole grains, unprocessed meats, and healthy fats, covering many nutrients patients ask about. Evidence shows the Mediterranean diet can reduce cardiovascular risk, type 2 diabetes, obesity, and cancer risk—including lung cancer—which are common COPD comorbidities. It also offers anti-inflammatory and fiber-related benefits, even if it is not a cure for COPD.
Due to different stages of COPD, the individualized approach really is key. On one hand patients in earlier stages or newly diagnosed benefit from the Mediterranean diet to help manage symptoms, slow progression, and address comorbidities like heart disease and diabetes. On the other hand patients with advanced disease may be malnourished and experiencing sarcopenia, which increases higher risk and mortality, according to experts affiliated with the American Lung Association. For these patients, the goal is to increase strength and energy using calorie-dense but healthy foods—such as full-fat dairy, olive oil, and nut butters—delivered in small, frequent meals to avoid breathlessness from stomach fullness.
According to Lung Cancer Research Fund, “lung cancer is the most common cancer worldwide. It is the most common cancer in men and the 2nd most common cancer in women” with “China, the US and Japan having the highest number of lung cancer cases in 2022.”[3] Obviously, it is more common in smokers than nonsmokers—about 80%–90% of lung cancer cases occur in people who are current or former smokers—yet lung cancer in never-smokers is still a significant public health issue and is associated with other risk factors such as second-hand smoke exposure, radon, air pollution, occupational exposures, and genetic susceptibility.
“Lung cancer in nonsmoking individuals accounts for 15% to 20% of lung cancer cases worldwide. Among patients with lung cancer, nonsmoking individuals are more likely to have genomic alterations such as EGFR mutations or ALK gene rearrangements, and these patients have improved survival when treated with tyrosine kinase inhibitors compared with chemotherapy.”[4]
While there are many synthetic alternatives as treatment, there are natural ones as well that show promising results in multiple cancer treatment studies. An animal study found that daily consumption of a kava-derived dietary supplement prevented the formation of 99 percent of tumors in a mouse lung tumor model, which was used to predict lung cancer in humans. Kava was also used in a pilot clinical trial among active smokers where it was proven to be effective in many aspects. “Kava increased urinary excretion of total NNAL [4-(Methylnitrosamino)-1-(3-pyridyl)-1-butanol] and reduced urinary 3-methyladenine in participants, suggestive of its ability to reduce the carcinogenicity of NNK [nicotine-derived nitrosamine ketone]. Kava also reduced urinary total nicotine equivalents, indicative of its potential to facilitate tobacco cessation. Plasma cortisol and urinary total cortisol equivalents were reduced upon kava use, which may contribute to reductions in tobacco use. These results demonstrate the potential of kava intake to reduce lung cancer risk among smokers.”[5]
Supporting our natural healing ability
The immune system relies on specialized T lymphocyte subsets to defend against pathogens while maintaining tolerance to self-antigens. The thymus is central to this process, producing T cells which enable broad antigen recognition and play a key role in immune surveillance against malignancies. “Though the thymus has the capacity to self-regenerate, it is often insufficient to reconstitute an intact thymic function. Thymic dysfunction leads to an increased risk of opportunistic infections, tumor relapse, autoimmunity, and adverse clinical outcome. Thus, exploiting the mechanism of thymic regeneration would provide new therapeutic options for these settings.”[6]
Support for the thymus gland can be achieved through a healthy diet, sleep patterns—so that the body produces melatonin and receives adequate rest—water, exercise and our learning to manage stress effectively. Glandular therapy (Thymus Gland) could be a great alternative as well which supports the diseased or weaken glands of the patient with reciprocal healthy and young animal glands rich in specific therapeutic nutrients. Glandular therapy is an effective medicine which uses animal tissue in order to provide the missing hormones, polypeptide enzymes, amino acids, and nutrients to the specific organs in need.
There is an alternative that works with your body with lifestyle changes: exercise with an organic diet and USP quality holistic natural medication as nature has intended.
The products that are helpful for winter:
- DMSO for NAMs and pain control and useful in prevent tissue freezing,
- Thymus Gland for increased T-cells for immunity,
- Kava Kava for preventing lung cancer,
- Antifungal Medication for fungal infections,
- fortified Boron and Colloidal Silver for immunity
We must be vigilant as we are fighting to remain healthy one step at a time.
References:
- Chen, Hanbiao et al. 2025. The effect of commonly used non-antibiotic medications on antimicrobial resistance development in Escherichia coli. https://www.nature.com/articles/s44259-025-00144-w
- Duah, Maxwell et al. 2021. Thymus Degeneration and Regeneration. https://pmc.ncbi.nlm.nih.gov/articles/PMC8442952/
- Lung Cancer Research Fund. 2026.
- Murphy, Cian et al. 2025. Lung Cancer in Nonsmoking Individuals. A Review. https://jamanetwork.com/journals/jama/fullarticle/2840343?guestAccessKey=a488770a-9a12-477a-b1e9-b719cf63a0a4&utm_medium=email&utm_source=postup_jn&utm_campaign=article_alert-jama&utm_content=etoc-tfl_&utm_term=112525
- Pierard, Pascale. 2025. Common Painkillers May Accelerate Antibiotic Resistance, Study Warns. https://www.medscape.com/viewarticle/common-painkillers-may-accelerate-antibiotic-resistance-2025a1000rvy
- Wang, Yi et al. 2020. The Impact of One-week Dietary Supplementation with Kava on Biomarkers of Tobacco Use and Nitrosamine-based Carcinogenesis Risk among Active Smokers. https://pubmed.ncbi.nlm.nih.gov/32102948/
[1] Chen, Hanbiao et al. 2025.
[2] Pierard, Pascale. 2025.
[3] Lung Cancer Research Fund. 2026.
[4] Murphy, Cian et al. 2025.
[5] Wang, Yi et al. 2020.
[6] Duah, Maxwell et al. 2021.
